IJMS | Free Full-Text | Hyperoside Induces Breast Cancer Cells Apoptosis via ROS-Mediated NF-κB Signaling Pathway | HTML
ROS scavenger, N‐acetyl‐l‐cysteine and NOX specific inhibitor, VAS2870 reduce platelets apoptosis while enhancing their viability during storage - Hosseini - 2019 - Transfusion - Wiley Online Library
Effects of N-acetyl cysteine on mitochondrial ROS, mitochondrial dynamics, and inflammation on lipopolysaccharide-treated human apical papilla cells | SpringerLink
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N-acetylcysteine attenuates PM2.5-induced apoptosis by ROS-mediated Nrf2 pathway in human embryonic stem cells - ScienceDirect
Effect of the reactive oxygen species (ROS) inhibitor N-acetylcysteine... | Download Scientific Diagram
ROS inhibitor NAC compromises cytotoxicity of ADM/DAC treatment in HCC-LM3 and SMMC-7721 cell lines but does not impact glucose metabolism which is depressed by DAC.
Cytochrome P450-dependent reactive oxygen species (ROS) production contributes to Mn 3 O 4 nanoparticle-caused liver injury - RSC Advances (RSC Publishing) DOI:10.1039/C8RA05633A
Regulation of Reactive Oxygen Species by Atm Is Essential for Proper Response to DNA Double-Strand Breaks in Lymphocytes | The Journal of Immunology
N-acetylcysteine attenuates lipopolysaccharide-induced impairment in lamination of Ctip2-and Tbr1- expressing cortical neurons in the developing rat fetal brain | Scientific Reports
Antioxidant treatment induces reductive stress associated with mitochondrial dysfunction in adipocytes - Journal of Biological Chemistry
The mechanism of action of N-acetylcysteine (NAC): The emerging role of H2S and sulfane sulfur species - ScienceDirect
N-Acetylcysteine amide, a Thiol Antioxidant and Neuroprotective Agent, Reduces ROS Production - Immune System Research
N-Acetyl Cysteine (NAC)-Directed Detoxification of Methacryloxylethyl Cetyl Ammonium Chloride (DMAE-CB) | PLOS ONE
Reactive oxygen species mediate soft corals-derived sinuleptolide-indu | OTT
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N-Acetyl Cysteine Depletes Reactive Oxygen Species and Prevents Dental Monomer-Induced Intrinsic Mitochondrial Apoptosis In Vitro in Human Dental Pulp Cells | PLOS ONE
Figure 5 from Effect of N-acetyl-L-cysteine on ROS production and cell death caused by HEMA in human primary gingival fibroblasts. | Semantic Scholar
![N-acetylcysteine alleviates PCB52-induced hepatotoxicity by repressing oxidative stress and inflammatory responses [PeerJ]](https://dfzljdn9uc3pi.cloudfront.net/2020/9720/1/fig-5-full.png "N-acetylcysteine alleviates PCB52-induced hepatotoxicity by repressing oxidative stress and inflammatory responses [PeerJ]")
N-acetylcysteine alleviates PCB52-induced hepatotoxicity by repressing oxidative stress and inflammatory responses [PeerJ]
Curcumin induced oxidative stress attenuation by N-acetylcysteine co-treatment: a fibroblast and epithelial cell in-vitro study in idiopathic pulmonary fibrosis | Molecular Medicine | Full Text
Importance of ROS-mediated autophagy in determining apoptotic cell death induced by physapubescin B - ScienceDirect
